Surgical removal of endometriotic lesions alters local and systemic proinflammatory cytokines in endometriosis patients

Friday, April 1, 2016
Excising endometriotic lesions significantly decreased circulating levels of inflammatory cytokines. Levels were also higher than in normal women, dissimilar in eutopic versus ectopic endometrium, and influenced by disease stage.

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Granulocyte colony stimulating factor prevents loss of spermatogenesis after sterilizing busulfan chemotherapy

Monday, December 29, 2014
Spermatogenesis can be destroyed by alkylating chemotherapies. In this study, spermatogenesis and undifferentiated spermatogonia in mice treated with busulfan were protected by treatment with granulocyte colony-stimulating factor.

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Follicular phase ovarian follicular fluid and plasma cytokine profiling of natural cycle in vitro fertilization patients

Wednesday, July 30, 2014
Follicular fluid cytokine concentrations vary throughout the follicular phase in the natural cycle and demonstrate complex interrelationships that underpin normal folliculogenesis and subsequent oocyte and embryo development.

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Urinary cytokine and chemokine profiles across the menstrual cycle in healthy reproductive age women

Wednesday, April 30, 2014
Using longitudinal data on menstrual cycle function, we evaluated urinary cytokines levels for assessment of reproductive function by considering detection, variation across the menstrual cycle, and correlations with hormones.

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The disturbance of TH17 Treg cell balance in adenomyosis

Friday, January 31, 2014
The disturbance of TH17-Treg cell balance was involved in the immunopathogenesis of both diffuse and focal forms of adenomyosis and showed positive correlation with dysmenorrhea severity and CA-125 levels.

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Coculturing human endometrial epithelial cells and stromal fibroblasts alters cell specific gene expression and cytokine production

Monday, September 30, 2013
A coculture system of polarized human endometrial epithelium and subject-paired stroma reveals effects of paracrine signaling on cell-specific global gene expression and cytokine production.

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Prolactin and proinflammatory cytokine expression at the fetomaternal interface in first trimester miscarriage

Friday, June 28, 2013
Impaired prolactin (PRL) expression in samples from miscarriages and the particular expression of the proinflammatory cytokines suggest the pivotal role of PRL in vital pregnancies, reciprocally affected by interleukin-2 expression.

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Proinflammatory cytokines induced altered expression of cyclooxygenase-2 gene results in unreceptive endometrium in women with idiopathic recurrent spontaneous miscarriage

Wednesday, January 2, 2013
Aberrant expression of proinflammatory, anti-inflammatory, and angiogenic cytokines alters the endometrial milieu, affecting the endometrial development and receptivity during implantation window in women with idiopathic recurrent spontaneous miscarriage.

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Interleukin 19 and Interleukin 22 serum levels are decreased in ovarian endometrioma

Wednesday, January 2, 2013
Serum interleukin-19 and -22 are decreased in ovarian endometrioma. They may be involved in the pathogenesis of endometriosis.

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Inflammation: a link between endometriosis and preterm birth

Wednesday, June 6, 2012
Inflammatory events are involved in the pathogenesis of endometriosis and preterm birth, and a clinical correlation is suggested.

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Low-molecular-weight heparins induce decidual heparin-binding epidermal growth factor–like growth factor expression and promote survival of decidual cells undergoing apoptosis

Friday, June 1, 2012
The effects of tinzaparin and enoxaparin on decidual cells were evaluated. Tinzaparin induced heparinbinding epidermal growth factor–like growth factor expression and secretion. Both low-molecular-weight heparins prevented tumor necrosis factor (TNF) a–induced apoptosis of decidual cells.

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Retinoic acid suppresses growth of lesions, inhibits peritoneal cytokine secretion, and promotes macrophage differentiation in an immunocompetent mouse model of endometriosis

Thursday, May 31, 2012
Retinoic acid inhibition of endometriotic-implant development in mouse model endometriosis may be related to suppression of interleukin-6 and macrophage chemotactic factor-1 production, and promotion of peritoneal macrophage differentiation.

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